« Previous
Next »
Clinical Neurophysiology
Volume 121, Issue 4
, Pages 459-460
, April 2010
Copper toxicity in the general population
References
- . Treatment of Wilson’s disease with zinc: XV long-term follow-up studies. J Lab Clin Med. 1998;132(4):264–278
- . Copper proof: chronic copper toxicity and the epidemics of Alzheimer’s disease and dementia. Ann Arbor, MI: George J. Brewer, Inc.; 2009;
- . Treatment of Wilson’s disease with zinc. II. Validation of oral 64 copper with copper balance. Am J Med Sci. 1986;292(6):344–349
- . Dietary copper and high saturated and trans fat intakes associated with cognitive decline. Arch Neurol. 2006;63(8):1085–1088
- Is cognitive function linked to serum free copper levels? A cohort study in a normal population. Clin Neurophysiol. 2010;121(4):502–507
- . Trace amounts of copper in water induce beta-amyloid plaques and learning deficits in a rabbit model of Alzheimer’s disease. Proc Natl Acad Sci USA. 2003;100(19):11065–11069
- . Trace copper levels in the drinking water, but not zinc or aluminum, influence CNS Alzheimer-like pathology. J Nutr Health Aging. 2006;10(4):247–254
- . Excess of serum copper not related to ceruloplasmin in Alzheimer disease. Neurology. 2005;64(6):1040–1046
- . Excess of nonceruloplasmin serum copper in AD correlates with MMSE, CSF [beta]-amyloid, and h-tau. Neurology. 2006;67(1):76–82
- . Longitudinal prognostic value of serum “free” copper in patients with Alzheimer disease. Neurology. 2009;72(1):50–55
PII: S1388-2457(09)00784-6
doi: 10.1016/j.clinph.2009.12.015
© 2009 International Federation of Clinical Neurophysiology. Published by Elsevier Inc. All rights reserved.
« Previous
Next »
Clinical Neurophysiology
Volume 121, Issue 4
, Pages 459-460
, April 2010

