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Volume 120, Issue 1 , Pages 3-10 , January 2009

Characteristics of generalised epileptiform activity

H. Aurlien
- Section of Clinical Neurophysiology, Department of Neurology, Haukeland University Hospital, Jonas Liesvei 65, N-5021 Bergen, Norway
- Corresponding author. Tel.: +47 55975000; fax: +47 55975164.
I.O. Gjerde
- Department of Neurology, Haukeland University Hospital, Bergen, Norway
G.E. Eide
- Centre for Clinical Research, Haukeland University Hospital, Bergen, Norway
- Department of Public Health and Primary Health Care, University of Bergen, Bergen, Norway
J.C. Brøgger
- Section of Clinical Neurophysiology, Department of Neurology, Haukeland University Hospital, Jonas Liesvei 65, N-5021 Bergen, Norway
- Department of Clinical Medicine, University of Bergen, Bergen, Norway
N.E. Gilhus
- Department of Neurology, Haukeland University Hospital, Bergen, Norway
- Department of Clinical Medicine, University of Bergen, Bergen, Norway

,Accepted 15 October 2008.

Age distribution at test time for 325 consecutive GEA patients.

Age distribution at test time for 325 consecutive GEA patients.
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Fitted values for AR-amplitude (a), AR-frequency (b), GBA-amplitude (c), and GBA-frequency (d) by age in 325 GEA patients and in 3268 drug-free outpatient controls. Shaded areas are fitted values ±1.9

Fitted values for AR-amplitude (a), AR-frequency (b), GBA-amplitude (c), and GBA-frequency (d) by age in 325 GEA patients and in 3268 drug-free outpatient controls. Shaded areas are fitted values ±1.96 SE.
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Pairwise correlation (Spearman’s) between AR-amplitude, AR frequency, GBA-amplitude, and GBA-frequency in 325 consecutive GEA patients (red) and 3268 controls (green). Each background activity variabl

Pairwise correlation (Spearman’s) between AR-amplitude, AR frequency, GBA-amplitude, and GBA-frequency in 325 consecutive GEA patients (red) and 3268 controls (green). Each background activity variable is plotted against the other ones. Histograms show the number of EEGs with identified AR/GBA-amplitude/frequency in EEGs with GEA and in controls. The black lines in the histograms indicate the normal distribution.
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Fitted values for GEA-amplitude (a) and GEA-frequency (b) by age in 325 GEA patients. Shaded areas are fitted values ±1.96 SE.

Fitted values for GEA-amplitude (a) and GEA-frequency (b) by age in 325 GEA patients. Shaded areas are fitted values ±1.96 SE.
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Age-related predicted probability for PPR in 259 GEA patients provoked with flicker stimulation. The predicted probabilities are adjusted for significant predictors such as ‘polyspike slow wave’, ‘pol

Age-related predicted probability for PPR in 259 GEA patients provoked with flicker stimulation. The predicted probabilities are adjusted for significant predictors such as ‘polyspike slow wave’, ‘polyspikes’ ‘gender’, and ‘medicated’. Shaded areas are predicted values ±1.96 SE.
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Predicted probability for RBS GEA by age in 325 GBA patients. Shaded areas are predicted values ±1.96 SE.

Predicted probability for RBS GEA by age in 325 GBA patients. Shaded areas are predicted values ±1.96 SE.
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Age-related probability of GEA-types in 325 consecutive GEA patients.

Age-related probability of GEA-types in 325 consecutive GEA patients.
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Age-related probability of current brain-related disease except epilepsy (CBRDEE) in 325 consecutive GEA patients.

Age-related probability of current brain-related disease except epilepsy (CBRDEE) in 325 consecutive GEA patients.
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